[Severe hypomagnesemia refractory to oral supplementation associated to omeprazole treatment].

chronic venous thromboses were also more common than acute thrombosis. Squizzato et al.1 reported 13 cases of acute cerebral sinus venous thrombosis associated with thyroid disease. The most common etiology of hyperthyroidism was Graves--Basedow disease, and a hypercoagulability state and even vascular compression in the event of large goiter were suggested as a pathogenetic mechanism. In the reported case, autoimmune hyperthyroidism could have been Hashimoto toxicosis instead of Graves--Basedow disease. Both of them may show diffuse hyperuptake at gammagraphy, but the absence of positive TSH receptor antibodies and the good response to antithyroid treatment suggest Hashimoto toxicosis as the first possibility, despite 2. Siegert CEH, Smelt AHM, De Bruin TWA. Superior sagittal sinus thrombosis and thyrotoxicosis. Possible association in two cases. Stroke. 1995;26:496--7. 3. Verberne HJ, Fliers E, Prummel FM, Stam J, Brandjes PD, Wiersinga WM. Thyrotoxicosis as a predisposing factor for cerebral venous thrombosis. Thyroid. 2000;10: 607--10. 4. Mouton S, Nighoghossian N, Berruyer M, Derex L, Philippeau F, Cakmak S, et al. Hyperthyroidism and cerebral venous thrombosis. Eur Neurol. 2005;54:78--80. 5. Strada L, Gandolfo C, Del Sette M. Cerebral sinus venous thrombosis in a subject with thyrotoxicosis and MTHFR gene polymorphism. Neurol Sci. 2008;29:343--5. 6. Franchini M, Lippi G, Targher G. Hyperthyroidism and venous thrombosis: a casual or causal association? A systematic literature review. Clin Appl Thromb Hemost. 2011;17: escargado de http://www.elsevier.es el 05/04/2017. Copia para uso personal, se prohíbe la transmisión de este documento por cualquier medio o formato.